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Advances in Hydrodechlorination Technologies for Diclofenac Removal from Aqueous Systems.

水系中のジクロフェナク除去を目的とした接触水素脱塩素化技術の最新動向

review not specified not assessed

Abstract

This review consolidates current knowledge on the catalytic hydrodechlorination (HDC) of diclofenac (DFC), a widely detected pharmaceutical contaminant in water systems. The survey covers the principal catalytic materials employed for DFC removal from aqueous matrices, evaluating their performance characteristics. Three strategic approaches are examined: conventional HDC relying on molecular hydrogen as the electron donor, biocatalytic methods, and electrocatalytic alternatives. Optimized reaction conditions for each system are outlined alongside kinetic models and mechanistic frameworks that clarify the HDC process. The review concludes by identifying future directions for catalyst development and potential contributions to wastewater remediation and heterogeneous catalysis research.

Mechanism

HDC proceeds via reductive cleavage of C–Cl bonds in diclofenac at the catalyst surface, using molecular hydrogen as the electron donor; biocatalytic and electrocatalytic routes achieve analogous dechlorination through alternative electron-transfer mechanisms.

Bibliographic

Authors
Castillo C, Mora JA, Brijaldo MH
Journal
Molecules
Year
2025 (2025-08-09)
PMID
40871486
DOI
10.3390/molecules30163332
PMC
PMC12388549

Tags

Disease:腸管障害 Mechanism:抗酸化酵素 活性酸素種

Delivery context

The delivery route is not clearly identifiable from this paper. For hydrogen intake, inhalation is the most efficient route; inhalation, however, carries explosion risk (empirical LFL of 10%; high-concentration devices are not recommended).

Safety notes

The delivery route is not clearly identifiable from this paper. For hydrogen intake, inhalation is the most efficient route; inhalation, however, carries explosion risk (empirical LFL of 10%; high-concentration devices are not recommended).

See also:

Other papers on the same disease / condition

Cite as: H2 Papers — PMID 40871486. https://h2-papers.org/en/papers/40871486
Source: PubMed PMID 40871486